Tss(Tail-short Shionogi), a New Short Tail Mutation Found in the BALB/cMs Strain, Maps Quite Closely to the Tail-short(Ts) Locus on Mouse Chromosome 11.
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A short note on the tail bound of Wishart distribution
We study the tail bound of the emperical covariance of multivariate normal distribution. Following the work of (Gittens & Tropp, 2011), we provide a tail bound with a small constant. 1 Main result Let {ξk : k = 1 · · ·n} follow multivariate normal distribution Nd(0, C). The scatter matrix S = ∑n k=1 ξkξ > k follows Wishart distribution, Wd(n,C). The estimate of C is 1 nS. The tail bound of S ha...
متن کاملThe X-linked mouse mutation Bent tail is associated with a deletion of the Zic3 locus.
Bent tail (BN:) is a spontaneous, semi-dominant mutation on the mouse X chromosome that produces tail deformities and, rarely, open neural tube defects. Analysis of 292 normal male and affected male and female progeny from an intraspecific back-cross involving BN: supports a gene order of cen-DXMit89-18.5 +/- 2.3 cM-DXMit166-1.4 +/- 0.7 cM-BN:-1.0 +/- 0.6 cM-DXMit140 -4.8 +/- 1.3 cM-DXBay6-tel....
متن کاملGenetical studies on the skeleton of the mouse. XXII. The development of Danforth's short-tail.
T H E semi-dominant gene for Danforth's short-tail in the mouse (symbol Sd; linkage group V) was first described by Dunn, Gluecksohn-Schoenheimer, & Bryson (1940). The most conspicuous abnormality of Sdl + heterozygotes is a shortening of the tail the extent of which varies with the genetic background (Dunn, 1942; Fisher & Holt, 1944; Dunn & Gluecksohn-Schoenheimer, 1945). Reduction or absence ...
متن کامل"Bent-Tail," A Dominant, Sex-Linked Mutation in the Mouse.
Although sex-linkage in animals is no longer an unusual phenomenon, its apparent rarity in rodents is noteworthy, especially in the intensively studied house mouse and rat. Hauschka, et al.,1 have presented evidence for a sex-linked lethal altering the sex ratio in the house mouse; Falconer2 recently reported a second sex-linked mutation in the house mouse. A third sex-linked mutation, Bent-tai...
متن کاملPinX1 the tail on the chromosome.
The PinX1 protein inhibits telomerase, an enzyme that lengthens telomeres - the structures that protect the ends of chromosomes. Loss of PinX1 leads to increased telomere length along with defects in chromosome dynamics. In this issue of the JCI, Zhou et al. present novel evidence from human tumors and mouse models indicating that PinX1 is a clinically significant tumor suppressor. Importantly,...
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ژورنال
عنوان ژورنال: Experimental Animals
سال: 2000
ISSN: 1341-1357,1881-7122
DOI: 10.1538/expanim.49.131